Study Suggests Krill Oil , Could Protect Against , Certain Hallmarks of Aging.
PsyPost reports that a recent study out of Norway found that krill oil can be used to protect neurons from age-related degeneration.
According to the study published in 'Aging,' researchers found that krill oil rewires gene expression that contributes to a number of age-related chemical changes in the cell.
According to the study published in 'Aging,' researchers found that krill oil rewires gene expression that contributes to a number of age-related chemical changes in the cell.
Krill, a type of small crustacean, are found across the oceans of the world.
Krill, a type of small crustacean, are found across the oceans of the world.
Extracts from a certain species of Antarctic krill have a high content of long-chain omega-3 fatty acids.
These fatty acids are important in the brain with diverse roles ranging from maintenance of brain structure and function and serve as essential building blocks of healthy cell membranes.
, Via the 'Krill oil protects dopaminergic neurons from age-related degeneration through temporal transcriptome rewiring and suppression of several hallmarks of aging' study.
The study focused on the effects of krill oil on dopaminergic neurons.
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PsyPost reports that these neurons are located in the midbrain and are responsible for producing the neurotransmitter dopamine.
The loss of these neurons has been linked with the development of Parkinson’s disease, one of the most prominent neurological disorders aging humans face.
According to the team's work, krill oil is capable of rewiring distinct gene expression programs that contribute to several hallmarks of aging.
According to the team's work, krill oil is capable of rewiring distinct gene expression programs that contribute to several hallmarks of aging.
PsyPost reports that while the study makes an important contribution to the field of biochemistry of aging, it remains to be seen if the effects differ in human trials.
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PsyPost reports that while the study makes an important contribution to the field of biochemistry of aging, it remains to be seen if the effects differ in human trials.
